Search results for "ERK pathway"

showing 10 items of 192 documents

The Role of Phospholipase D and MAPK Signaling Cascades in the Adaption of Lichen Microalgae to Desiccation: Changes in Membrane Lipids and Phosphopr…

2016

Classically, lichen phycobionts are described as poikilohydric organisms able to undergo desiccation due to the constitutive presence of molecular protection mechanisms. However, little is known about the induction of cellular responses in lichen phycobionts during drying. The analysis of the lipid composition of the desiccated lichen microalga Asterochloris erici revealed the unusual accumulation of highly polar lipids (oligogalactolipids and phosphatidylinositol), which prevents the fusion of membranes during stress, but also the active degradation of cone-shaped lipids (monogalactosyldiacylglycerol and phosphatidylethanolamine) to stabilize membranes in desiccated cells. The level of pho…

0106 biological sciences0301 basic medicineMAPK/ERK pathwayLichensPhysiologyMAP Kinase Signaling SystemMembrane lipidsPlant ScienceBiology01 natural sciencesDesiccation toleranceDephosphorylation03 medical and health scienceschemistry.chemical_compoundMembrane LipidsChlorophytaOsmotic PressureMicroalgaePhospholipase DPhosphorylationProtein kinase ADehydrationPhospholipase DKinaseCell BiologyGeneral MedicinePhosphatidic acidPhosphoproteinsAdaptation Physiological030104 developmental biologychemistryBiochemistrylipids (amino acids peptides and proteins)010606 plant biology & botanyPlantcell physiology
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Nitric Oxide in Plants: Production and Cross-talk with Ca2+ Signaling

2008

International audience; Nitric oxide (NO) is a diatomic gas that performs crucial functions in a wide array of physiological processes in animals. The past several years have revealed much about its roles in plants. It is well established that NO is synthesized from nitrite by nitrate reductase (NR) and via chemical pathways. There is increasing evidence for the occurrence of an alternative pathway in which NO production is catalysed from L-arginine by a so far non-identified enzyme. Contradictory results have been reported regarding the respective involvement of these enzymes in specific physiological conditions. Although much remains to be proved, we assume that these inconsistencies can …

0106 biological sciencesMAPK/ERK pathwayArabidopsisPlant ScienceCalcium-Transporting ATPasesBiologyNitrate reductaseArginine01 natural sciencesPlant Physiological PhenomenaNitrate ReductaseNitric oxide03 medical and health scienceschemistry.chemical_compoundNitrateProtein kinasesNitrilesAnimals[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyNitriteMolecular BiologyNitritesPlant Physiological Phenomena030304 developmental biologyMammals0303 health sciencesKinasefungiNitric oxidechemistryBiochemistrySecond messenger systemCitrullineCalciumCryptogeinNitric Oxide SynthaseGenome Plant010606 plant biology & botanySignal Transduction
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Activation of a nuclear-localized SIPK in tobacco cells challenged by cryptogein, an elicitor of plant defence reactions.

2009

When a plant cell is challenged by a well-defined stimulus, complex signal transduction pathways are activated to promote the modulation of specific sets of genes and eventually to develop adaptive responses. In this context, protein phosphorylation plays a fundamental role through the activation of multiple protein kinase families. Although the involvement of protein kinases at the plasma membrane and cytosolic levels are now well-documented, their nuclear counterparts are still poorly investigated. In the field of plant defence reactions, no known study has yet reported the activation of a nuclear protein kinase and/or its nuclear activity in plant cells, although some protein kinases, e.…

0106 biological sciencesMAPK/ERK pathwayMolecular Sequence DataActive Transport Cell NucleusBiology01 natural sciencesBiochemistryMAP2K703 medical and health sciencesCytosolTobaccoASK1Protein phosphorylation[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyAmino Acid SequenceNuclear proteinProtein kinase AMolecular BiologyConserved Sequence030304 developmental biologyPlant ProteinsCell Nucleus0303 health sciencesKinasePlant ExtractsAlgal ProteinsLife SciencesCell BiologyCell biologyEnzyme ActivationBiochemistrySignal transductionMitogen-Activated Protein KinasesSequence Alignment010606 plant biology & botanySignal TransductionThe Biochemical journal
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AM fungal exudates activate MAP kinases in plant cells in dependence from cytosolic Ca2+ increase

2011

International audience; The molecular dialogue occurring prior to direct contact between the fungal and plant partners of arbuscular-mycorrhizal (AM) symbioses begins with the release of fungal elicitors, so far only partially identified chemically, which can activate specific signaling pathways in the host plant. We show here that the activation of MAPK is also induced by exudates of germinating spores of Gigaspora margarita in cultured cells of the non-leguminous species tobacco (Nicotiana tabacum), as well as in those of the model legume Lotus japonicus. MAPK activity peaked about 15 min after the exposure of the host cells to the fungal exudates (FE). FE were also responsible for a rapi…

0106 biological sciencesMAPK/ERK pathwayTime FactorsMAP Kinase Signaling SystemPhysiologyNicotiana tabacumLotus japonicusPlant ScienceComplex MixturesBiology01 natural sciences03 medical and health sciencesPlant CellsTobaccoBotanyGenetics[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyGlomeromycotaSymbiosisNicotiana plumbaginifoliaPlant Proteins030304 developmental biologyMitogen-Activated Protein Kinase Kinasesarbuscular-mycorrhizal fungi0303 health sciencesdiffusible factorcalciumKinasefungiArbuscular-mycorrhizal fungi; Signaling; Diffusible factor; MAPK; Calciumfood and beveragesSpores FungalPlant cellbiology.organism_classificationMAPKsym pathwayCell biologyCytosolCell cultureLotus[SDE.BE]Environmental Sciences/Biodiversity and Ecologysignaling010606 plant biology & botanyPlant Physiology and Biochemistry
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Integrated signaling network involving calcium, nitric oxide, active oxygen species but not mitogen-activated protein kinases in BcPG1-elicited grape…

2006

We have already reported the identification of the endopolygalacturonase 1 (BcPG1) from Botrytis cinerea as a potent elicitor of defense responses in grapevine, independently of its enzymatic activity. The aim of the present study is the analysis of the signaling pathways triggered by BcPG1 in grapevine cells. Our data indicate that BcPG1 induces a Ca2+ entry from the apoplasm, which triggers a phosphorylation-dependent nitric oxide (NO) production via an enzyme probably related to a NO synthase. Then NO is involved in i) cytosolic calcium homeostasis, by activating Ca2+ release from internal stores and regulating Ca2+ fluxes across the plasma membrane, ii) plasma membrane potential variat…

0106 biological sciencesMAPK/ERK pathwayTime FactorsPhysiology[SDV]Life Sciences [q-bio]Phenylalanine ammonia-lyase01 natural sciencesNitric oxideFungal Proteins03 medical and health scienceschemistry.chemical_compounddepolarizationGene Expression Regulation Plantplant defensenitric oxideVitisdépolarisationProtein kinase ACells Cultured030304 developmental biology0303 health scienceselicitorbiologyelicitor; grapevine; plant defense; nitric oxideKinaseGeneral MedicinePlants Genetically ModifiedElicitorgrapevinechemistryBiochemistryMitogen-activated protein kinasebiology.proteinCalciumBotrytisMitogen-Activated Protein KinasesSignal transductionbotrytis cinereavigneReactive Oxygen SpeciesAgronomy and Crop ScienceSignal Transduction010606 plant biology & botany
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Desensitization of cAMP Accumulation via Human β3-Adrenoceptors Expressed in Human Embryonic Kidney Cells by Full, Partial, and Biased Agonists

2019

β3-Adrenoceptors couple not only to cAMP formation but, at least in some cell types, also to alternative signaling pathways such as phosphorylation of extracellular signal-regulated kinase (ERK). β3-Adrenoceptor agonists are used in long-term symptomatic treatment of the overactive bladder syndrome; it is only poorly understood which signaling pathway mediates the clinical response and whether it undergoes agonist-induced desensitization. Therefore, we used human embryonic kidney cells stably transfected with human β3-adrenoceptors to compare coupling of ligands with various degrees of efficacy, including biased agonists, to cAMP formation and ERK phosphorylation, particularly regarding des…

0301 basic medicineAgonistMAPK/ERK pathwaymedicine.drug_classmedicine.medical_treatmentdesensitization03 medical and health scienceschemistry.chemical_compoundpartial agonism0302 clinical medicinecAMPIsoprenalinemedicinePharmacology (medical)β3-adrenoceptorOriginal ResearchDesensitization (medicine)PharmacologyForskolinKinaselcsh:RM1-950extracellular signal-related kinaseCell biologylcsh:Therapeutics. Pharmacology030104 developmental biologybiased agonismchemistry030220 oncology & carcinogenesisPhosphorylationSignal transductionmedicine.drugFrontiers in Pharmacology
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Integrated analysis of colorectal cancer microRNA datasets: Identification of microRNAs associated with tumor development

2018

Colorectal cancer (CRC) is one of the leading cause of cancer death worldwide. Currently, no effective early diagnostic biomarkers are available for colorectal carcinoma. Therefore, there is a need to discover new molecules able to identify pre-cancerous lesions. Recently, microRNAs (miRNAs) have been associated with the onset of specific pathologies, thus the identification of miRNAs associated to colorectal cancer may be used to detect this pathology at early stages. On these bases, the expression levels of miRNAs were analyzed to compare the miRNAs expression levels of colorectal cancer samples and normal tissues in several miRNA datasets. This analysis revealed a group of 19 differentia…

0301 basic medicineMAPK/ERK pathwayAgingColorectal cancerDatasets as TopicBiology03 medical and health sciences0302 clinical medicineMismatch Repair PathwaymicroRNAmedicineHumansSettore MED/05 - Patologia ClinicaGenePI3K/AKT/mTOR pathwayBioinformaticWnt signaling pathwayMicroRNAbioinformaticsBiomarkerCell Biologymedicine.diseaseColorectal cancerBiomarker (cell)MicroRNAs030104 developmental biology030220 oncology & carcinogenesisBioinformatics; Biomarker; Colorectal cancer; Dataset; MicroRNA; Aging; Cell BiologyCancer researchColorectal NeoplasmsTranscriptomeResearch PaperDataset
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Targeting BRAF and RAS in Colorectal Cancer

2021

Simple Summary In colorectal cancer, mutations of the KRAS and BRAF genes are quite common and can contribute to the activation of cell signaling pathways that lead to cell proliferation and differentiation. These processes promote cancer growth, and in some cases, they may cause cells to develop resistance to certain types of treatment, notably EGFR inhibitors. We summarize recent knowledge regarding the effects of KRAS and BRAF mutations in the setting of colorectal cancer and discuss the new therapies under development. Abstract Colorectal cancer (CRC) is still one of the most frequent forms of cancer in the world in terms of incidence. Around 40% of CRC patients carry a mutation of the …

0301 basic medicineMAPK/ERK pathwayCancer ResearchColorectal cancerAngiogenesismedicine.medical_treatmentcolorectal cancerReviewmedicine.disease_causeBRAFTargeted therapy03 medical and health sciences0302 clinical medicineKRASmedicineneoplasmsRC254-282EGFR inhibitorsMutationbusiness.industryNeoplasms. Tumors. Oncology. Including cancer and carcinogensCancertargeted therapymedicine.diseasedigestive system diseases030104 developmental biologyOncology030220 oncology & carcinogenesisCancer researchKRASbusinessCancers
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CXCR7 Reactivates ERK Signaling to Promote Resistance to EGFR Kinase Inhibitors in NSCLC

2019

Abstract Although EGFR mutant–selective tyrosine kinase inhibitors (TKI) are clinically effective, acquired resistance can occur by reactivating ERK. We show using in vitro models of acquired EGFR TKI resistance with a mesenchymal phenotype that CXCR7, an atypical G protein-coupled receptor, activates the MAPK–ERK pathway via β-arrestin. Depletion of CXCR7 inhibited the MAPK pathway, significantly attenuated EGFR TKI resistance, and resulted in mesenchymal-to-epithelial transition. CXCR7 overexpression was essential in reactivation of ERK1/2 for the generation of EGFR TKI–resistant persister cells. Many patients with non–small cell lung cancer (NSCLC) harboring an EGFR kinase domain mutatio…

0301 basic medicineMAPK/ERK pathwayCancer ResearchLung NeoplasmsDrug ResistanceDrug resistanceTransgenicMiceChemokine receptor0302 clinical medicineNeoplasmsCarcinoma Non-Small-Cell LungReceptorsMedicineNon-Small-Cell LungCXCRReceptorLungbeta-ArrestinsCancerEGFR inhibitorsTumorKinaseLung CancerErbB ReceptorsOncology5.1 Pharmaceuticals030220 oncology & carcinogenesisDevelopment of treatments and therapeutic interventionsTyrosine kinaseEpithelial-Mesenchymal TransitionMAP Kinase Signaling SystemOncology and CarcinogenesisMice TransgenicArticleCell LineExperimental03 medical and health sciencesClinical ResearchCell Line TumorAnimalsHumansOncology & CarcinogenesisProtein Kinase InhibitorsReceptors CXCRbusiness.industryCarcinomaNeoplasms Experimentalrespiratory tract diseases030104 developmental biologyProtein kinase domainDrug Resistance NeoplasmMutationCancer researchNeoplasmbusinessCancer Research
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Noncanonical GLI1 signaling promotes stemness features and in vivo growth in lung adenocarcinoma

2016

Aberrant Hedgehog/GLI signaling has been implicated in a diverse spectrum of human cancers, but its role in lung adenocarcinoma (LAC) is still under debate. We show that the downstream effector of the Hedgehog pathway, GLI1, is expressed in 76% of LACs, but in roughly half of these tumors, the canonical pathway activator, Smoothened, is expressed at low levels, possibly owing to epigenetic silencing. In LAC cells including the cancer stem cell compartment, we show that GLI1 is activated noncanonically by MAPK/ERK signaling. Different mechanisms can trigger the MAPK/ERK/GLI1 cascade including KRAS mutation and stimulation of NRP2 by VEGF produced by the cancer cells themselves in an autocrin…

0301 basic medicineMAPK/ERK pathwayCancer ResearchLung NeoplasmsPyridinesPyridineMitogen-Activated Protein Kinase KinaseMice SCIDMiceCarcinoma Non-Small-Cell LungRNA Small InterferingNon-Small-Cell LungMolecular Biology; Genetics; Cancer ResearchTumorbiologyintegumentary systemHedgehog signaling pathwayCell biologyNeoplastic Stem CellsFemaleRNA InterferenceOriginal ArticleHumanXenograft Model Antitumor AssayAdenocarcinomaSCIDSmall InterferingZinc Finger Protein GLI1Cell LineProto-Oncogene Proteins p21(ras)Adenocarcinoma; Animals; Carcinoma Non-Small-Cell Lung; Cell Line Tumor; Female; Humans; Lung Neoplasms; Mice; Mice SCID; Mitogen-Activated Protein Kinase Kinases; Neoplastic Stem Cells; Neuropilin-2; Proto-Oncogene Proteins p21(ras); Pyridines; Pyrimidines; RNA Interference; RNA Small Interfering; Xenograft Model Antitumor Assays; Zinc Finger Protein GLI1; Molecular Biology; Genetics; Cancer Research03 medical and health sciencesParacrine signallingGeneticSettore MED/04 - PATOLOGIA GENERALEstem cellsCancer stem cellGLI1Cell Line TumorGeneticsAnimalsHumansAutocrine signallingMolecular BiologyMitogen-Activated Protein Kinase KinasesSettore MED/06 - ONCOLOGIA MEDICAAnimalCarcinomaXenograft Model Antitumor AssaysNeuropilin-2Lung Neoplasmlung cancer030104 developmental biologyPyrimidinesPyrimidineCancer cellbiology.proteinRNANeoplastic Stem CellSmoothened
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